its due to DNa repair mechanism and some genetic abnormalities in
certain metabolisms.genetics affects ageing also.we ll see
how.normally in DNa replication 5 end of daughter DNa is not formeeway
the usual enzyme DNa polymerase.so such an end strand is called
telomere.its prone to be destroyed easily.so its prevented by
telomerase.its a reverse transcriptase enzyme..its a DNa dependantrna polymerase.
so in old people this enzyme is very less so cells are destroyed
quickly.in cancer cells is is abundant.so cells keep proliferating.
in werners syndrome,telomerase is absent.so a 20 years young man will
appear like an 80 years old man.it is called as cellular senescen

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